DIRECTIONS | CONTACT US |
Drug-induced nonimmune thrombocytopenia
HOME > Encyclopedia > Drug-induced nonimmune thrombocytopenia
Print | Email

Drug-induced nonimmune thrombocytopenia

Definition

Drug-induced nonimmune thrombocytopenia is a condition in which certain drugs reduce the number of normal clot-forming blood cells (platelets).

Alternative Names

Thrombocytopenia (nonimmune) drug-induced; Nonimmune thrombocytopenia - drug-induced

Causes

Platelets are important in forming blood clots. Certain drugs may decrease the number of platelets by damaging the bone marrow where platelets are made. Decreased platelets may cause easy bruising or abnormal bleeding.

Bleeding can be life-threatening if it occurs in the brain or other vital organs. Other drugs (such as aspirin) can increase the risk of bleeding by stopping platelets from working normally.


Review Date: 4/27/2007
Reviewed By: Rita Nanda, M.D., Section of Hematology/Oncology, University of Chicago Medical Center, Chicago, IL. Review provided by VeriMed Healthcare Network.
The information provided herein should not be used during any medical emergency or for the diagnosis or treatment of any medical condition. A licensed medical professional should be consulted for diagnosis and treatment of any and all medical conditions. Call 911 for all medical emergencies. Links to other sites are provided for information only -- they do not constitute endorsements of those other sites. © 1997- A.D.A.M., Inc. Any duplication or distribution of the information contained herein is strictly prohibited.
adam.com
About Us | Contact Us | Patient and Visitors Guide | Request an Appointment | Calendar | Gift Shop

The University of Tennessee Medical Center | 1924 Alcoa Highway Knoxville, Tennessee 37920-6969 | Telephone: 1.865.305.9000.
To make an appointment, call 1.877.UT.CARES (1.877.882.2737) Privacy | SiteMap | Employee Access

The University of Tennessee Medical Center provides medical treatment without regard to disability, age, race, color, religion, sex or national origin.